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Larger context for LACK in Corpus USbrown_UKbncw/US_brown.txt

A reason for such wide variation in the pulmonary morphology is entirely lacking at present.
Within certain wide limits anatomy dictates function and, if one is permitted to speculate, potential pathology should be included in this statement as well.
For example, the marked susceptibility of the monkey to respiratory infection might be related to its delicate, long alveolar ducts and short, large bronchioles situated within a parenchyma entirely lacking in protective supportive tissue barriers such as those found in types 1, and 3,.
One might also wonder if monkeys are capable of developing bronchiolitis as we know it in man or the horse.
In addition, it would be difficult to imagine chronic generalized emphysema occurring in a cow, considering its marked lobular development but, conversely, not difficult to imagine this occurring in the horse or the dog.
Anatomically, the horse lung appears to be remarkably like that of man, insofar as this can be ascertained from comparison of our findings in the horse with those of others (Birnbaum, '54) in the human being.
The only area in which one might find major disagreement in this matter is in regard to the alveolar distribution of the bronchial arteries.
As early as 1858, Le Fort claimed an alveolar distribution of the bronchial arteries in human beings.
In 1951, this was reaffirmed by Cudkowicz.